What to do if you have hiccups due to cerebral infarction_What to do if you have hiccups due to cerebral infarction

Patients with cerebral infarction have relatively poor specific body functions. For example, many patients with cerebral infarction have poor gastric motility, so they will inevitably have symptoms of indigestion. In this case, patients with cerebral infarction are more likely to experience hiccups. After hiccups caused by cerebral infarction, in addition to taking care of the stomach, you must also actively treat the cause, that is, treat cerebral infarction. Below, we will introduce in detail the solutions to hiccups caused by cerebral infarction and the treatment methods for cerebral infarction.

1. What to do if you have hiccups due to cerebral infarction

The common cause of hiccups in patients with cerebral infarction is poor gastric motility, causing functional dyspepsia or hiccups caused by vagus nerve excitement. The treatment for the former is mainly to eat small meals frequently, mainly soft and easily digestible foods, get out of bed and move around more, massage the abdomen, and symptomatic treatments such as itopride hydrochloride and Weifuan can be given. The latter can also choose symptomatic treatments such as itopride hydrochloride and Weifuan.

2. Treatment of cerebral infarction

1. General treatment in the acute phase

During the acute phase, patients should rest in bed as much as possible and strengthen the care of the skin, oral cavity, respiratory tract, bowel and urination. Pay attention to the balance of water and electrolytes. If patients are still unable to eat on their own 48 to 72 hours after onset, they should be given nasogastric liquid diet to ensure nutritional supply. The patient's daily care, diet, and treatment of other complications should be given priority. In addition, most patients, their relatives and some medical staff hope that there will be better medicines to help patients recover soon, but ignore other aspects of treatment, such as the patient's diet. Since some patients with cerebral infarction cannot take care of themselves and even have difficulty swallowing during the acute phase, problems with their metabolism will quickly arise if they are not given proper nutrition and energy. At this time, even the best treatment medications will hardly produce good results.

2. Treatment of cerebral edema

(1) Mannitol: 20% mannitol hypertonic solution is commonly used in clinical practice. Mannitol is one of the most commonly used and effective dehydrating agents.

(2) 10% glycerol fructose can produce pharmacological effects through hypertonic dehydration. It can also utilize the energy generated by glycerol metabolism into the brain metabolism process, thereby improving local metabolism. Through the above effects, it can reduce intracranial pressure and intraocular pressure, eliminate cerebral edema, increase cerebral blood volume and cerebral oxygen consumption, and improve brain metabolism.

(3) Diuretic dehydration agents such as furosemide (Lasix) and sodium urate can be injected intermittently intramuscularly or intravenously.

(4) Adrenal cortical hormones are mainly glucocorticoids such as hydrocortisone and cortisone. Their secretion and production are regulated by adrenocorticotropic hormone (ACTH), which has anti-inflammatory, immunosuppressive and anti-shock effects.

(5) Human albumin (albumin) Human albumin is a medium molecular weight colloid that plays an important role in generating colloid osmotic pressure, which helps retain fluid in the vascular cavity.

3. Acute thrombolytic therapy

Thrombosis and embolism are the basis of cerebral infarction, so the ideal method is to restore normal blood flow to ischemic brain tissue before necrosis occurs. Early reperfusion of brain tissue with cerebral blood flow can reduce the degree of ischemia and limit the damage to nerve cells and their functions.