Is there any scientific basis for low carbohydrates to prevent colorectal cancer?

Is there any scientific basis for low carbohydrates to prevent colorectal cancer? It is reported that although the occurrence of colorectal cancer is related to a carbohydrate-rich Western diet, the detailed mechanism is still unclear. For more information on this issue, please see the following:

A study published in the journal Cell on July 17 shows that the metabolism of dietary carbohydrates by gut microbes promotes the proliferation of intestinal cells and the formation of tumors in mice with a genetic predisposition to colorectal cancer. Treatment with antibiotics or a low-carbohydrate diet significantly reduced the incidence of colorectal cancer in mice with a genetic predisposition to colorectal cancer, suggesting that perhaps simple measures (antibiotic treatment or a low-carbohydrate diet) can also be used to prevent colorectal cancer in humans in the future.
"It is critical to understand how environmental factors, such as gut microbes and diet, interact with genetic factors to influence the progression of colorectal cancer," said Alberto Martin, a researcher at the University of Toronto. The new study shows that gut bacteria interact with a carbohydrate-rich diet to trigger a common type of hereditary colorectal cancer. In a Western diet, carbohydrates make up about half of an adult's daily calorie intake, and previous studies have found that a carbohydrate-rich diet has been associated with the development of colorectal cancer. Colorectal cancer is also frequently associated with mutations in the tumor suppressor gene APC and the MSH2 gene, which is known to play a key role in repairing DNA damage.
However, researchers have not been clear why mutations in pathways involved in DNA repair (APC and MSH2 genes) are more common in colorectal cancer than in other cancers. Because gut microbes are also involved in the development of colorectal cancer, Martin and his team suspected that these mutations might interact with diet to further promote the progression of this type of cancer.