Sequelae of traumatic cerebral infarction

Cerebral infarction is a relatively serious disease that can affect the entire body. In severe cases, the patient will be unable to take care of himself. There are many types of cerebral infarction, and traumatic cerebral infarction is relatively common. This phenomenon is caused by trauma to the brain without good treatment. Traumatic cerebral infarction is more likely to cause various sequelae. Next, let’s take a look at the sequelae of traumatic cerebral infarction.

Mild sequelae of cerebral infarction

For mild cerebral infarction, in this case, you need to consider taking medication for prevention, such as aspirin enteric-coated tablets, Buchang Naoxintong capsules and other drugs for conditioning treatment, regular check-ups, and diet, try to eat light food, and avoid greasy food to avoid aggravating symptoms.

Sequelae of cerebral infarction Coronary heart disease Coronary artery disease

In case of coronary heart disease after cerebral infarction, we should pay attention to low-fat and low-salt diet, control the cause, and give aspirin enteric-coated tablets, lovastatin, and Tongxinluo drug treatment.

Sequelae of cerebral infarction: twitching of the corner of the mouth, inflexibility of the right hand

In this case, you need to go to the hospital's TCM department for treatment and choose a reasonable Chinese medicine prescription through syndrome differentiation and treatment.

Right occipital lobe infarction

Traumatic cerebral infarction Cerebral infarction caused by head trauma is called traumatic cerebral infarction. This disease is more common in adolescents, all of whom have a history of head trauma, and neurological localization signs often appear within 24 hours after the injury. It is less common for symptoms to appear 2 weeks or immediately after the injury. Cerebral angiography, CT or MRI can help confirm the diagnosis. Cerebral infarction caused by head trauma is called traumatic cerebral infarction. This disease is more common in adolescents, all of whom have a history of head trauma, and neurological localization signs often appear within 24 hours after the injury. It is less common for symptoms to appear 2 weeks or immediately after the injury. Cerebral angiography, CT or MRI can help confirm the diagnosis. In recent years, due to the widespread use of CT, the diagnosis rate of traumatic cerebral infarction in children has been significantly improved. Its pathogenesis is related to arterial intima damage and vascular spasm. In the event of head trauma, the sudden extension and flexion of the head and neck will cause traction on the blood vessels in the neck, causing contusion of the blood vessel walls or damage to the endothelium. On the one hand, it will directly form traumatic thrombus; on the other hand, it can reflexively cause vasospasm. Vasospasm itself provides the potential for thrombosis. At the same time, because of ischemic changes caused by vascular injury, spasm, or thrombosis, the damaged blood vessel wall can become a site for delayed thrombosis. The thrombus expands or breaks off, blocking the basilar artery or posterior cerebral artery, thereby causing cerebral infarction. In addition, traumatic cerebral infarction may also be associated with the formation of dissecting aneurysms. Due to traumatic damage, the impact of blood flow between the inner and middle layers of cerebral blood vessels causes progressive separation, forming a dissecting aneurysm, progressive narrowing of the blood vessel lumen, and ultimately leading to vascular occlusion. However, due to the incomplete development of the brain and physiological and anatomical characteristics of children such as thin blood vessels, even a slight traumatic blow can cause occlusion of deep intracranial perforating branches and branch vessels, resulting in cerebral infarction.