Uremia can actually cause heart failure, so the treatment of uremia should certainly be taken seriously. Common treatments include limiting water and sodium intake, correctly using some diuretics, and drinking plenty of water to help reduce the burden on the heart. 1. Limit water and sodium intake. 2. Use of diuretics: Generally, fast-acting and powerful diuretics are used in large dosages to increase the excretion of water and sodium and reduce the burden on the heart. 3. Digitalis: It is advisable to use digitalis drugs with a short half-life and use them in reduced doses according to creatinine clearance rate. 4. Vasodilators: They are particularly effective for patients with congestive heart failure with low cardiac output and those who have poor responses to diuretics and digitalis, especially those with significant hypertension. In addition, nitrate drugs can be used to increase venous capacity, reduce the amount of blood returning to the heart, lower the diastolic filling pressure of the heart, and reduce pulmonary congestion. The causes of heart failure caused by uremia are as follows: 1. The role of uremic toxins. In uremia, harmful metabolites accumulate in the body, and toxins inhibit the myocardium, causing cardiomyopathy, leading to myocardial dysfunction and heart failure. 2. Patients with uremia have excessive blood volume. In uremia, blood volume increases, and the left ventricular end-diastolic volume, cardiac output and cardiac output increase. When cardiac function cannot compensate, left ventricular dysfunction may occur and lead to heart failure. 3. Renal anemia in patients with uremia. Long-term anemia in uremia patients causes myocardial hypoxia and decreased myocardial function. As the body compensates, the heart rate increases and cardiac output increases. Over time, the heart may fail due to excessive load and myocardial hypoxia. |
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