30-year-old mild cerebral infarction

Cerebral infarction is more likely to occur after the age of 50. Many people's bodies gradually deteriorate when they are 30 years old, and they are also prone to cerebral infarction. Don't worry about mild cerebral infarction. You can recover by paying attention to conditioning exercises. Because people at the age of 30 are still very young, they will not be too weak like middle-aged and elderly people, so recovery at this time will be stronger. Many diseases that occur when they are young will be better recovered.

Disease diagnosis

1. Cerebral hemorrhage usually occurs during activities or when people are emotionally excited. Most patients have a history of hypertension and their blood pressure fluctuates greatly. The onset is acute, and headaches, vomiting, and impaired consciousness are common. High-density hemorrhage foci can be seen on brain CT scans.

2. Brain tumors: Slowly progressive cerebral infarction should be differentiated from brain tumors. Primary brain tumors develop slowly, and the onset of brain metastases is sometimes similar to acute cerebrovascular disease. A brain CT scan should be performed in time. If a brain tumor cannot be differentiated from cerebral infarction, it is best to do a brain MRI to confirm the diagnosis.

Inspection method

Laboratory tests:

1. Cerebrospinal fluid examination Cerebrospinal fluid examination is generally not performed at present, and cerebrospinal fluid examination is not used as a routine examination for ischemic cerebrovascular disease. The cerebrospinal fluid of most patients with cerebral infarction is normal. However, the pressure may increase in patients with a large infarction area and obvious cerebral edema. A small number of patients with hemorrhagic infarction may have an increase in red blood cells, and white blood cells and cell phagocytosis may occur in the later stages.

2. Routine blood, urine and stool tests and biochemical examinations are mainly related to risk factors for cerebrovascular disease such as hypertension, diabetes, hyperlipidemia, heart disease, and atherosclerosis.

Other auxiliary examinations:

1. Brain CT scan The main manifestations of brain CT scan for cerebral infarction are: ① Low density of lesions: This is an important characteristic manifestation of cerebral infarction, and this sign may be caused by ischemic edema of brain tissue. ② Local brain tissue swelling: manifested as disappearance of cerebral sulci, compression and deformation of cerebral cisterns and ventricles, and displacement of midline structures to the contralateral side, that is, brain CT scan shows a space-occupying effect. This sign can be observed 4 to 6 hours after onset. ③Dense arterial shadow: The density of the main cerebral arteries is increased, which is common in the middle cerebral artery. The mechanism of occurrence is that the thrombus or embolus stands out because of its higher density than the contralateral or surrounding brain tissue. Some patients may experience symptoms within 24 hours of ischemia.

2. Brain MRI examination can detect cerebral infarction at an early stage, especially lesions in the brainstem and cerebellum. The T1 and T2 relaxation times are prolonged. On weighted images, T1 shows low signal in the lesion area and T2 shows high signal. Brain MRI examination can detect smaller infarction lesions, and brain MRI diffusion imaging can reflect new infarction lesions.

MRI has shown its advantages in the evaluation of early diagnosis and differential diagnosis of ischemic cerebral infarction. In recent years, superconducting high-end magnetic resonance equipment has been put into clinical use. The application of magnetic resonance diffusion-weighted imaging (DWI) and perfusion-weighted imaging (PWI) based on planar echo (EPI) technology has made certain progress in the early diagnosis of cerebral infarction and even in the study of the correlation between blood perfusion changes in the acute cerebral infarction area and pathophysiological processes.