What are the chemotherapy options for nasopharyngeal carcinoma? Which mechanism is better?

What are the chemotherapy options for nasopharyngeal carcinoma? Which mechanism is better?

Nasopharyngeal carcinoma is a disturbing disease that cannot be treated blindly. In addition to medical and drug treatment, nursing work is particularly important. Patients who maintain an optimistic attitude are conducive to your recovery and treatment process. The author introduces below, what is the chemotherapy plan for nasopharyngeal carcinoma? Which mechanism is good?

What are the chemotherapy options for nasopharyngeal carcinoma?

Treatment options include:

⑴CBF regimen: 600~10000 mg of cyclophosphamide per time, intravenous injection, applied on days 1 and 4. Bleomycin 15 mg per time, intramuscular injection, applied on days 1 and 5. 5-fluorouracil 5000 mg, intravenous injection, applied on days 2 and 5. Rest after the treatment course, 4 courses per week in total. The efficiency is 60.8%.

(2) PFA regimen: 20 mg of cisplatin and 500 mg of 5-fluorouracil, intravenous drip for 5 days; 40 mg of doxorubicin, intravenous injection on the first day of the treatment. Repeat once after 3 to 4 weeks, which can significantly reduce the tumor.

⑶PF regimen: cisplatin 20mg/m2 and 5-fluorouracil 500mg/m2, intravenous drip, continuous use for 5 days and then rest for 2 weeks, 2 to 3 courses of treatment. This regimen can be used to shrink tumors before radiotherapy or for cases with simple chemotherapy, with an effective rate of 93.7%.

Which mechanism is better?

Nasopharyngeal carcinoma is highly malignant and prone to distant metastasis. In the process of actively seeking effective drugs, researchers have conducted a lot of detailed research and achieved quite effective results. Studies have shown that ginsenoside Rh has a wide range of anti-tumor mechanisms, mainly through regulating the proliferation cycle of tumor cells, inducing cell differentiation and apoptosis to exert anti-tumor effects.

Ginsenoside Rh2 can inhibit the growth of nasopharyngeal cancer cells and reduce or eliminate the volume of cancer tissue. Ginsenoside Rh2 induces tumor cells to differentiate into normal cells, which is beneficial to controlling tumor development, inducing tumor cell apoptosis, and forming apoptotic bodies after cell disintegration, without causing inflammatory reactions in surrounding tissues, solving the biggest swelling and pain problem of nasopharyngeal cancer patients.

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