Where is the most common site of colorectal cancer?

Where is the most common site of colorectal cancer? Colorectal cancer is the most common malignant tumor disease of the digestive system. It has a very high incidence rate in recent years, mainly due to changes in people's eating habits and an increase in bad eating habits. So where is the most common site of colorectal cancer? Let's take a look.

The most common sites of colorectal cancer are: rectum, sigmoid colon, cecum, ascending colon, and descending colon. Since benign tumors such as intestinal polyps are precancerous lesions, the most common sites of colorectal polyps are also the most common sites of colorectal cancer. A large number of clinical observations have found that 50% of cancers occur in the rectum and 25% in the sigmoid colon.

The main function of the large intestine is to absorb water from food residues, gradually forming them into feces and excreting them from the body. As food residues move through the large intestine, water is gradually absorbed, and by the time they reach the sigmoid colon, feces have been formed. At this time, the harmful substances and carcinogens in the feces are also highly concentrated. If they cannot be excreted from the body in time, under the action of some bacteria in the intestine (such as anaerobic bacteria), they will cause adverse stimulation to the intestinal mucosa over time, causing congestion, exudation, edema and other inflammatory reactions to damage the intestinal mucosa. In the process of repairing the damage, some genes of intestinal epithelial cells may mutate, become defective, and lose their normal functions, causing atypical hyperplasia and tumor-like hyperplasia of the intestinal epithelium, and eventually malignant transformation to become colorectal cancer.

The second is the right ileocecal region. The reason for this is related to the function of the large intestine. The main function of the large intestine is to absorb water from food residues, gradually form them, and excrete them into feces. In the process of repairing damage to intestinal epithelial cells, some genes may mutate, become defective, and lose their normal functions, causing atypical hyperplasia and tumor-like hyperplasia of the intestinal epithelium, and eventually become malignant and become colorectal cancer.