What is the cause of bite pain

If you feel pain when biting your teeth, you may have problems with the tooth joints. For example, you may have eaten something hard recently, or often drink cold drinks. These irritants can cause tooth pain. More serious problems are that biting is a stimulation to the healthy function of the tooth tissue, so it can cause diseases around the teeth, such as periodontitis, destruction of the periodontal tissue, damage to the periodontal tissue, etc.

What to do if your teeth clench and hurt?

Pain when biting teeth is usually caused by apical periodontitis. Clinical symptoms include a feeling of swelling in the teeth, pain when biting, and the affected teeth making contact first when biting.

If the treatment is not timely or handled improperly, periapical abscess may occur. The periapical abscess breaks through the tympanic membrane and reaches the submucosal membrane, and a sense of fluctuation can be smelled. Oral anti-inflammatory drugs such as penicillin, broad-spectrum antibiotics such as amorin, or first- and second-generation cephalosporin tablets can be taken. , and metronidazole tablets can also be used.

If the pain is obvious, you can take high potassium tablets or ibuprofen capsules orally. Taking medicine only relieves temporary symptoms. Tooth treatment is the most important thing. Under local anesthesia, you can open the pulp, expand the root canal, remove the residual necrotic tissue in the root canal, keep the root canal open, and place pain-relieving cotton twists at the root canal opening to facilitate drainage. Then, after the symptoms disappear, root filling and root canal treatment can be performed.

Can periodontal disease cause biting pain?

When periodontal disease is severe, many patients will experience biting pain. This is because the normal bite force itself is a functional stimulation to the healthy periodontal tissue, which can maintain the normal metabolism of the periodontal tissue.

If periodontal disease occurs, the supporting tissues around the teeth are damaged, and the ability of the periodontal tissues to withstand bite forces decreases. At this time, normal bite forces can also cause damage to the periodontal tissues, which manifests as bite pain and bite trauma. The main factor causing bite trauma is insufficient periodontal support. After periodontal disease, the periodontal supporting tissues are absorbed by the alveolar bone, and the fibers of the periodontal membrane become loose or reduced, which reduces their supporting strength. Normal bite forces can cause bite pain.

1. Temporomandibular joint pain

Many people do not have much idea about joint pain, because they do not have the disease themselves and do not know what symptoms will occur. The causes and manifestations are also different. There are many kinds of joint pain. For example, this product mainly comes from psychological factors. Because of mental tension and excessive anxiety, the pain will be aggravated. In addition, it is not ruled out that it is due to immune system problems, dental functional problems, and joint overload.

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It is generally believed to be related to the following factors:

1. Psychosocial factors: Patients often have mental symptoms such as anxiety, irritability, mental tension, excitability and insomnia. Some patients can clearly have a causal relationship between mental and emotional factors and the onset of the disease. In chronic and protracted patients, the influence of mental factors on the recurrence of symptoms can also be found.

2. Occlusal factors: Clinical examination of TMD patients often reveals obvious occlusal factors, including interference, premature cusp contact, severe lockbite, deep overbite, majority of posterior teeth missing, and excessive wear of the occlusal surfaces resulting in a low vertical distance.

3. Immune factors: Immunological studies have shown that the main components of articular cartilage, such as collagen polysaccharides and chondrocytes, are antigenic. Because articular cartilage is wrapped in matrix, it is isolated from the vascular system from embryo to adult, becoming a closed antigen that cannot be recognized by the body's immune system. Related experimental studies have shown that TMD also involves cellular immunity.

4. Excessive load on joints: Moderate weight-bearing is necessary and important for maintaining the normal structure, function and physiological remodeling of joints. However, excessive weight bearing beyond physiological limits can cause degenerative changes or even damage to the joints.

5. Joint anatomical factors: From a functional point of view, the temporomandibular joint and jaw have become more flexible as humans evolve to adapt to more complex jaw movements for speech and facial expressions. Because of this, from an anatomical point of view, the corresponding joints, muscles and ligaments become significantly weakened, and the load-bearing capacity of the joints decreases. This increase in the type, flexibility, and range of joint motion in humans is a potential threat to the anatomically weakened temporomandibular joint.

6. Other factors: Cold stimulation of the joint area, muscle dysfunction caused by bad posture and affecting the normal position of the mandible and condyle are also factors that induce temporomandibular joint disorder.

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Patients often experience chronic pain in the unilateral or bilateral temporomandibular joints. The pain may radiate behind the ears, limit mouth opening, and cause local traction pain. There is often a crisp sound when the temporomandibular joint moves, and the pain is often aggravated by chewing hard objects, yawning, and cold.

Auxiliary examination:

① Plain X-ray films (Schuhler view and transpharyngeal lateral condylar film) can reveal changes in the joint space and bone quality, such as sclerosis, bone destruction and hyperplasia, and cystic changes.

②Arthrography and magnetic resonance imaging can reveal articular disc displacement, perforation, and changes in articular disc attachments.

③Arthroendoscopic examination can reveal early changes of the disease.