Epidural anesthesia nerve damage

Epidural anesthesia nerve damage

Epidural anesthesia nerve damage can be caused by a variety of reasons. After epidural anesthesia spinal nerve damage, the patient generally experiences pain in one lower limb, severe numbness, difficulty walking, unbearable pain, and even incontinence, and paralysis and weakness of both lower limbs. It can be seen how harmful this disease is. Therefore, once epidural anesthesia nerve damage is discovered, epidural filling therapy is recommended for treatment.

Epidural filling therapy is recommended, which injects the drug directly into the epidural space and allows it to diffuse and penetrate into the damaged spinal nerves and their surrounding areas. Reduce, eliminate edema, stabilize, excite, and repair affected nerve function in order to improve symptoms.

Many anesthesiologists are willing to induce paraesthesia in patients to localize the nerves when performing peripheral regional blocks. Although the presence of paresthesias suggests the presence of a needle stick injury and increases the risk of long-term postoperative paresthesias, there are no definitive data to confirm or refute this. Selander et al. showed that compared with the method of puncturing around blood vessels, the incidence of postoperative nerve injury increased when patients had paresthesia during axillary block, but the difference was not significant. Importantly, 40% of patients undergoing perivascular nerve blocks also experienced paresthesia, illustrating the difficulty in standardizing nerve localization and analyzing nerve injury.

Any regional block anesthetic can cause infection, but mononeurological sequelae are rare. The source of infection may be exogenous, such as contaminated medical devices or treatments; it may also be endogenous, such as secondary to infection near the puncture or catheterization site. Although infection at the puncture site is an absolute contraindication for regional block anesthesia, regional block anesthesia is often used for conditions such as infiltrative cellulitis, lymphangitis, and erythema. Indwelling catheters theoretically increase the risk of infection, but although colonization may occur, infection is rare. In case of local infection, the catheter can be removed and antibiotics can be used. The remaining catheter end can become a source of infection. The author has encountered a case of axillary abscess requiring surgical resection, but there were no neurological sequelae.

Neurologic deficits that develop within 24 hours after surgery suggest intra- or extra-neural hematoma, intra-neural edema, or involvement of a sufficient number of nerve fibers to allow prompt diagnosis. However, many of the delayed paresthesias after regional block anesthesia do not appear immediately after nerve damage, but often only appear after several days or weeks.

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