How long can you live with acidosis

Acidosis is usually caused by metabolic acidosis or respiratory acidosis. It can be detected by blood routine or electrocardiogram in time. During treatment, patients must rest in bed and keep warm. Once dehydration occurs, patients need to be injected with saline in time. Patients with severe conditions need to take alkaline drugs. So how long can acidosis last?

First, how long can you live with acidosis? Diabetic ketoacidosis is one of the most common acute complications of diabetes. The main symptoms include fatigue, nausea, vomiting, abdominal pain, and rapid heartbeat. In severe cases, dehydration and even coma may occur, which is life-threatening. Therefore, if you feel uncomfortable, you must go to the hospital in time. For mild cases, the symptoms will gradually improve after treatment such as fluid replacement, insulin therapy, and correction of electrolyte imbalance. If the condition is serious, or the patient is elderly and has cardiovascular and cerebrovascular diseases, the treatment process may be life-threatening. If diabetic ketoacidosis is treated effectively and aggressively, it will not be life-threatening after recovery.

Second, neonatal acidosis refers to the loss of balance in the newborn's internal environment. The causes of neonatal acidosis are: 1. Primary decrease in plasma and decreased pH value. 2. Respiratory nerve and muscle dysfunction. When acidosis occurs in a newborn, oxygen inhalation and dehydration treatment should be given promptly. For those with mild symptoms, it can be cured in 3-6 weeks; for those with more serious symptoms, the treatment time may need to be up to 3 months. The consequences of neonatal acidosis are relatively serious. Survival time is related to the quality of medical care and the baby's own physical condition. The recovery of each patient is different.

How long can a person live with acidosis? When a patient is in ketoacidosis, oxygen should be administered to prevent tissue hypoxia caused by the dissociation of oxygenated hemoglobin when correcting the acidosis. It may also be treated through diet. Most patients have obvious gastrointestinal symptoms and poor appetite, so easily digestible foods are mainly consumed. In ketoacidosis, the patient is in a coma and a gastric tube should be inserted to prevent vomiting and aspiration pneumonia. If urination is difficult, a urinary catheter should also be placed. Patients with hypotensive shock can be corrected after timely fluid replacement. If there is no response, plasma expanders should be given in time and vasoactive drugs should be given if necessary. Prolonged shock can lead to lactic acidosis.