When you hear about intracranial tumors, don't panic too much, because intracranial tumors are divided into benign and malignant tumors. When a benign tumor occurs, there is a great chance of recovery as long as the correct treatment is used. Therefore, after an intracranial tumor occurs in the human body, it is necessary to actively diagnose and find the correct method for treatment. What are the diagnostic methods for intracranial tumors? 1. Intracranial inflammation. Such as meningitis, arachnoiditis, brain abscess, intracranial inflammation generally has an acute or subacute onset, meningeal irritation signs and systemic symptoms. Optic disc edema is rare and mild in the early stages. Cerebrospinal fluid examination shows inflammatory manifestations and may detect pathogens. 2. Chronic subdural hematoma. It is usually seen in elderly people with head trauma, but sometimes the trauma is mild and they cannot recall it. The clinical manifestations may include mental symptoms similar to senile dementia, intracranial hypertension or impaired consciousness. Localized signs are mainly weakness of one side of the limbs. CT examination can confirm the diagnosis. 3. Cerebral cysticercosis. The patient may have stool worms or subcutaneous nodules, and often have epilepsy, mental symptoms or manifestations of intracranial hypertension. CT or MRI can detect lesions in the brain. 4. Epilepsy. Primary epilepsy usually begins before the age of 20 and has no localized neurological signs. Focal epilepsy that develops in adulthood is suspected to be an intracranial tumor. The patient may have intracranial hypertension and localized signs, and the tumor may be detected by imaging. 5. Multiple sclerosis. It is a common type of demyelination, characterized by diffuse axonal demyelination and gliosis. It often occurs around the ventricles, optic nerves, brainstem, cerebellar white matter and cerebellar peduncles, and spinal cord. Sometimes it needs to be differentiated from intracranial tumors, especially gliomas. Multiple sclerosis is common in young and middle-aged people, mostly women, and the course of the disease is characterized by alternating remissions and relapses. Imaging examinations showed that there were two or more lesions of varying ages in the white matter, most of which had no space-occupying effect. Active lesions can often be contrast-enhanced on CT or MRI, and steroid hormone treatment can reduce the enhancement density. |
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