Sulfuric acid is extremely acidic and will cause a burning sensation when dropped on the skin. If too much is used, it will cause burns. In this case, first wash with clean water, then apply magnesium oxide glycerin ointment to the affected area. If the effect is not obvious, be sure to seek medical attention in time. Experts remind people not to underestimate burns. If not treated in time, they may cause complications such as shock, sepsis, and acute renal failure. 1. What ointment should be used for sulfuric acid burns? The damage caused by concentrated sulfuric acid burns is extremely serious. The first urgent measure is to rinse with plenty of water and quickly take off clothes stained with sulfuric acid. Pay special attention to cleaning your eyes and mouth. Apply magnesium oxide glycerin ointment to the skin. If swallowed, rinse your mouth and drink plenty of water, take magnesium milk and seek medical attention immediately. What are the complications of burns? 1. Shock: In the early stage, it is mostly hypovolemic shock. If infection ensues, septic shock may occur. Severe burns may cause immediate shock due to the intense injury stimulation. 2. Sepsis: Burns cause defects in the skin's barrier function against bacteria; more serious patients also have weakened white blood cell function and immune function. Therefore, infection is prone to occur. The pathogenic bacteria are bacteria that commonly exist on the skin (such as Staphylococcus aureus, etc.) or bacteria that are exogenously contaminated (such as Pseudomonas aeruginosa, etc.). Purulent infection may occur on the wound surface and under the eschar. The infection may also develop into sepsis and septic shock. In addition, fungal infections may occur after the use of broad-spectrum antibiotics, especially in patients with systemic debilitation. 3. Pulmonary infection and acute respiratory failure: Pulmonary infection may have many causes, such as respiratory mucosal burns, pulmonary edema, atelectasis, sepsis, etc. Adult respiratory distress syndrome or pulmonary infarction may also occur, leading to acute respiratory failure. 4. Acute renal failure: There is renal ischemia before and after the concurrent shock. In severe cases, the renal capsule and renal tubules degenerate. In addition, hemoglobin, myoglobin, infectious toxins, etc. can damage the kidneys, thus leading to acute renal failure. 5. Stress ulcer and gastric dilatation: Erosion, ulcer and bleeding of the duodenal mucosa after burns are called Curling ulcers, which may be related to the ischemia of the gastrointestinal tract and the damage to the mucosa by hydrogen ion reflux after reperfusion. Gastric dilatation is often caused by the patient drinking a lot of water due to thirst when gastric motility is weakened in the early stage. 6. Others: Myocardial function is reduced and stroke volume may be reduced, which is related to the production of myocardial inhibitory factors, infectious toxins or myocardial hypoxia after burns. Brain edema or liver necrosis are also related to hypoxia, infection and toxins. It is worth noting that death from burns is often caused by multi-system organ failure. |
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