What are the clinical manifestations of cardiovascular neurosis

Cardiac neurosis is a syndrome centered around some symptoms of cardiovascular disease. Cardiovascular neurosis is more common among young and middle-aged people, and the incidence rate is higher in female patients than in male patients.

Cardiovascular neurosis is a clinical syndrome characterized by symptoms related to cardiovascular disease and is a type of functional neurosis. It mostly occurs in middle-aged and young people, and is more common between the ages of 20 and 50; it is more common in women than in men, especially menopausal women. There is no clinical evidence of organic heart disease and the prognosis is good, but patients with severe long-term symptoms may have significant impact on their normal life and work. Cardiovascular neurosis is a clinical syndrome characterized by symptoms related to cardiovascular disease and is a type of neurosis. Most cases occur in young and middle-aged people, with the highest incidence in those aged 20-40 years. The cause of the disease is still unclear, and may be related to nerve type, environmental factors, personality, genetics, etc.

Clinical manifestations

The main symptoms are numerous and changeable. They are generally subjective feelings, lack objective evidence, and there is no internal connection between the symptoms. The following cardiovascular disease symptoms are usually the main ones, which may be accompanied by other neurotic symptoms, such as insomnia, nightmares, irritability, restlessness, loss of appetite, dizziness, tinnitus, etc.

1. Palpitations are characterized by a feeling of increased heart rate and panic, which often worsens when one is nervous or tired.

2. Difficulty breathing, chest tightness, difficulty breathing, often feeling that there is not enough air, so you have to open the window, or even ask for oxygen. Many patients often take deep breaths or sigh-like breathing movements to relieve symptoms, which leads to hyperventilation, causing respiratory alkali poisoning and making symptoms worse.

3. The location of pain in the precordial area is not fixed; the onset of pain is not related to physical activity and most of the time it occurs in a resting state; the nature of the pain is often described as needle-like or pulling-like; the duration varies, but is generally longer; sublingual nitroglycerin cannot relieve the pain or may take dozens of minutes to relieve the pain.

4. Symptoms of autonomic dysfunction include sweating, cold hands and feet, tremors in both hands, frequent urination, increased bowel movements or constipation, etc.

In contrast to the numerous symptoms, physical examination lacked positive signs of significant pathological significance. It can be found that the heart rate is increased, the heart sounds are enhanced, there may be short systolic murmurs or premature beats, the blood pressure is slightly increased, and the leg reflexes are more active. Cardiac X-ray examination showed no abnormalities. The electrocardiogram may show sinus tachycardia, sinus arrhythmia, atrial or ventricular premature beats, and nonspecific ST-T wave changes.