Sequelae of cerebellar infarction

Most patients who suffer from cerebral infarction will have sequelae, such as difficulty in movement or speech dysfunction, and more serious symptoms include crooked mouth and squinting eyes. This is because the brain is infected by the cerebral infarction, coupled with blockage by arteriosclerosis, so that part of the blood circulation is poor and the brain cannot circulate and metabolize normally, which will cause partial functional disorders. What are the manifestations of the sequelae of cerebellar infarction?

The concept of sequelae of cerebral infarction

Or crossed paralysis, crossed sensory impairment, external ophthalmoplegia, nystagmus, dysphasia, language disorder, memory loss, facial paralysis, dysphagia

Difficulty in swallowing, choking on food and water, ataxia, dizziness, headache, etc. The fundamental reason for the above symptoms is the presence of blood diseases such as high blood viscosity, high blood lipids, high blood pressure, high blood sugar, platelet aggregation, and vascular diseases such as atherosclerotic plaque formation inside the cerebral blood vessels. The combined effect of the two diseases results in the formation of blood clots that block the cerebral arteries, leading to local blood flow interruption in the brain and ischemia, hypoxia and necrosis of brain tissue. If the motor nervous system controlled by the cranial nerves is affected, corresponding sequelae such as hemiplegia and limb disability will occur; if the language center controlled by the cranial nerves is affected, it will lead to corresponding neurological symptoms such as language disorders or even aphasia.

Treatment of sequelae of cerebral infarction

If limb and language disorders still exist one year after the onset of cerebral infarction, it is considered a sequelae of cerebral infarction. 30% of cerebral infarction patients will have sequelae of varying degrees. In my country, 30%-40% of cerebral infarction patients face recurrence. The focus of treatment in the sequelae period is to prevent recurrence of cerebral infarction and improve symptoms.

Some patients are able to take medication consistently within one year of onset, but ignore reliable medication after one year; some patients think that as long as they keep exercising and control their diet, it doesn’t matter whether they take medication or not. Relying solely on exercise and diet adjustment is primary prevention, and is a disease prevention measure for people who have not yet suffered cerebral infarction but have risk factors.

For patients who have already suffered a cerebral infarction, in order to prevent recurrence and continue to improve symptoms so that the condition continues to develop in a positive direction, in addition to paying attention to a light diet, functional exercise, and controlling blood pressure and blood lipids, the most important thing is to use reliable medications to continuously prevent and treat the causes of cerebral infarction.

The five-year recurrence rate of cerebral infarction is over 30%. Once a recurrence occurs, the patient and his family will face a heavier mental and economic burden. Therefore, it is better to prevent it before it occurs than to treat it after it occurs. Cerebral infarction is a chronic disease, and it is more common in the elderly around 60 years old. The focus of medication selection, in addition to the targeted preventive and therapeutic effects on the causes of cerebral infarction such as arteriosclerosis, should also have the advantages of long-term effectiveness, safety, no drug resistance, reasonable dosage form and small dosage. This is the best secondary prevention medication suitable for long-term use by middle-aged and elderly patients with cerebral infarction. The clinical key drugs that can be developed for secondary prevention are the first choice medications for the recovery and sequelae periods of cerebral infarction.