Eyelid twitching is very common in life, and this condition also has professional medical terminology in clinical practice. Some people also call eyelid twitching eyelid twitching. If you have eyelid twitching for a long time, you must pay attention to it. It is likely caused by a disease, so you must deal with it as soon as possible. Once the cause is clear, you can prescribe the right medicine. So why does my lower left eyelid twitch frequently? 1. Blepharospasm This may be blepharospasm, an unexplained, involuntary spasm and twitching of the muscles in the facial nerve innervation area. It is more common in middle-aged and elderly people. It is a neurological disease that causes great mental and physical pain to patients and also greatly affects their appearance. It does not have the clonic twitching of the lower facial muscles. The duration of the spasm can be long or short, and the spasm is manifested by repeated involuntary and strong eye closure. Many patients with blepharospasm have endured pain for a considerable period of time before receiving a clear diagnosis and treatment, and timely treatment is often delayed due to misdiagnosis. 2. Primary Essential blepharospasm is involuntary eyelid closure due to spasmodic contraction of the orbicularis oculi muscle, which is often bilateral and progressive. 2/3 are women, and most develop the disease over the age of 60. The cause is unknown. The frequency and duration of spasms vary. In mild cases, the orbicularis oculi muscle has paroxysmal, frequent small twitches that do not affect eye opening. In severe cases, the twitching is obvious, making it difficult to open the eyes, affecting vision and causing functional blindness. In most patients, symptoms stabilize within 3 to 5 years. One third of patients have associated movement disorders such as Meige syndrome, essential tremor, or Parkinson's disease. Secondary blepharospasm caused by keratoconjunctivitis, trichiasis, and blepharitis should be excluded during diagnosis. 3. Treatment The drug and physical treatments for this disease include: clonazepam, Antan, acupuncture, transcutaneous facial nerve thermolysis, etc., but all have little effect. Surgical treatments include: myectomy of the orbicularis oculi and supercilii muscles combined with browplasty and palpebrae superioris muscle reinforcement, and selective facial nerve extraction combined with muscle stripping. However, the effects of these methods are not ideal. The side effects of the former include: forehead numbness and eyelid edema, and the latter may have serious complications of facial nerve paralysis, manifested as ptosis, lagophthalmos, corneal exposure, and ectropion. 50% of patients relapse after surgery. |
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