Potassium Chloride Sustained Release Tablets Instructions

In life, many patients often suffer from insufficient food intake, vomiting, severe diarrhea or some other bad symptoms. In this case, we usually choose to take some related medicines, such as potassium chloride sustained-release tablets and other similar medicines that have therapeutic effects on these symptoms. Not only that, this type of medicine can also treat frequent, multi-source premature beats or rapid arrhythmias caused by digitalis poisoning. Now let's take a look at the instructions for potassium chloride sustained-release tablets!

1. Potassium chloride sustained-release tablets

1. Treatment of hypokalemia caused by various reasons, such as insufficient food intake, vomiting, severe diarrhea, use of potassium-excreting diuretics, hypokalemic familial periodic paralysis, long-term use of sugar

Corticosteroids and supplementation of hypertonic glucose, etc.

2. Prevention of hypokalemia When patients have potassium loss, especially if hypokalemia is harmful to the patient (such as patients with digitalis), potassium salt supplementation is required, such as eating very little, severe or chronic diarrhea, long-term use of adrenal

Corticosteroids, potassium-losing nephropathy, and Bartte's syndrome, etc. 3. Digitalis poisoning causes frequent, multifocal premature beats or rapid arrhythmias.

2. Drug Type

Class A work-related injury medical insurance double cross

3. Classification by Application

Electrolyte balance medication

4. Properties

This product is a sugar-coated tablet, which appears white after removing the coating.

5. Usage and Dosage

Adults take 0.5g~1g (1~2 tablets) at a time, 2~4 times a day, after meals, and adjust the dose according to the condition. The maximum daily dose for adults is 6g (12 tablets).

Those with gastrointestinal reactions can use oral solution instead, dilute it in cold boiled water or beverages and take it orally.

VI. Adverse Reactions

1. Oral administration may occasionally cause gastrointestinal irritation symptoms, such as nausea, vomiting, throat discomfort, chest pain (esophageal irritation), abdominal pain,

Diarrhea, even peptic ulcer and bleeding. It is more likely to occur when the patient is on an empty stomach, taking large doses, or has existing gastrointestinal diseases.

2. Hyperkalemia. It is prone to occur when the patient overdoses or has existing renal impairment. Symptoms include weakness, fatigue, numbness of hands, feet, lips and mouth, unexplained anxiety, confusion, difficulty breathing,

Slow heart rate, arrhythmia, conduction block, and even cardiac arrest. The electrocardiogram showed high and sharp T waves, and gradually

The PR interval is now prolonged. The P wave disappears, the QRS wave becomes wider, and a sine wave appears. Once hyperkalemia occurs, it should be treated immediately.

(1) Immediately stop potassium supplementation and avoid potassium-containing diets, medications, and potassium-sparing diuretics.

(2) Intravenous infusion of high-concentration glucose injection and insulin to promote the entry of K+ into cells, 10%-25% glucose injection 300-500 ml per hour. Every 20g of glucose plus regular

Insulin 10 units.

(3) If there is metabolite acidosis, 5% sodium bicarbonate injection should be used immediately. If there is no acidosis, 11.2% sodium lactate injection can be used, especially for those with widened QRS waves.

(4) Use calcium supplements to counteract the cardiotoxicity of K+. When the electrocardiogram shows a lack of P waves, widened QRS waves, and arrhythmia,

When using digitalis drugs, 10 ml of 10% calcium gluconate injection can be given intravenously over 2 minutes and repeated at 2-minute intervals if necessary.

(5) Oral administration of potassium-lowering resins to block intestinal K+ absorption and promote intestinal K+ excretion.

(6) Severe hyperkalemia associated with renal failure. Hemodialysis or peritoneal dialysis can be performed, and hemodialysis is more effective and faster in removing K+.

(7) Use loop diuretics and supplement with normal saline if necessary.

7. Taboos

Hyperkalemia; patients with scanty urine output and urinary retention.

8. Notes

This product should be swallowed whole and not crushed. Use with caution in the following situations:

1. Metabolic acidosis is accompanied by oliguria;

2. People with weakened adrenal cortex function;

3. Acute and chronic renal failure;

4. Acute dehydration, which can cause decreased urine volume and reduced urinary K+ excretion in severe cases;

5. Familial periodic paralysis; hypokalemic paralysis should be supplemented with potassium, but hyperkalemic or normokalemic periodic paralysis must be differentiated;

6. Chronic or severe diarrhea can cause hypokalemia, but it can also cause dehydration and hyponatremia, causing prerenal oliguria;

7. Patients with gastrointestinal obstruction, chronic gastritis, ulcer disease, esophageal stenosis, diverticulum, intestinal tension deficiency, and ulcerative enteritis should not take oral potassium supplements, because potassium will increase the stimulation to the gastrointestinal tract and aggravate the condition;

8. Conduction block arrhythmias, especially when using digitalis drugs;

9. Extensive burns, muscle trauma, severe infection, within 24 hours after major surgery, and severe hemolysis, the above conditions themselves can cause hyperkalemia;

10. Congenital adrenal hyperplasia with insufficient mineralocorticoid secretion.

11. The following follow-up examinations are required during medication;

(1) Blood potassium;

(2) Electrocardiogram;

(3) blood magnesium, sodium, and calcium;

(4) Acid-base balance index;

(5) Renal function and urine volume.