Glutamyl transpeptidase 120

Glutamyl transpeptidase is related to the metabolic function of the liver. Modern young people are under great pressure and often stay up late. Some people develop bad habits, such as drinking. Poisoning or environmental pollution can cause high glutamyl transpeptidase. However, if the body suffers from hepatitis, it can also cause high glutamyl transpeptidase. Is glutamyl transpeptidase 120 high?

Is glutamyl transpeptidase 120 high?

Glutamyl transpeptidase 120 is relatively high. The normal glutamyl transpeptidase is 0-50U/L. It is best to go to the hospital for a digestive system B-ultrasound and hepatitis B two-pair-and-a-half examination to see if you have hepatitis or liver disease. Under normal circumstances, the index of 120 is not considered high and it doesn’t matter much. Just quit smoking and drinking and eat more cabbage, radish, etc.

What are the effects on patients during the progression of liver disease?

1. Acute hepatitis

The symptoms are sudden onset of fatigue, loss of appetite, nausea, vomiting, jaundice (yellowing of the conjunctiva of both eyes and the skin and mucous membranes of the whole body, yellow urine like strong tea), abdominal distension, loose stools, discomfort or tenderness in the liver area and other hepatitis symptoms.

Although the onset of this process is rapid, it is usually not life-threatening as long as it is discovered and treated in time. For patients, the biggest impact is often due to gastrointestinal symptoms affecting work, life and study.

2. Chronic Hepatitis

Chronic onset, repeated attacks, and course of disease for more than half a year. Depending on the severity of the disease, it is divided into mild, moderate and severe. Mild patients have no or mild symptoms; moderate patients are between mild and severe; severe patients have obvious and persistent hepatitis symptoms.

The main harm of chronic hepatitis is that most patients are ignored because their symptoms are not obvious, resulting in continuous liver damage and progression to cirrhosis.

3. Hepatitis, cirrhosis, and liver cancer

Cirrhosis is the result of the progression of chronic hepatitis, and liver cancer is mostly formed on the basis of cirrhosis.

Patients with early cirrhosis have mild hepatitis symptoms, may have portal hypertension, mild esophageal varices, but no obvious signs of decompensation; as cirrhosis continues to progress, obvious liver failure occurs.

4. Liver failure

Acute hepatitis can progress to acute or subacute liver failure; acute onset may lead to acute-on-chronic liver failure on the basis of chronic hepatitis; chronic liver failure may occur on the basis of cirrhosis.

Liver failure is caused by severe damage to the liver, which leads to serious disorders or decompensation of its synthesis, metabolism, detoxification, excretion, and conversion functions, resulting in a series of serious and even life-threatening symptoms.

1. Extreme fatigue, with obvious anorexia, nausea, vomiting and other severe gastrointestinal symptoms.

2. Jaundice progressively worsens, and even "chole-enzyme dissociation" occurs, that is, the transaminase decreases, but the jaundice worsens.

3. Coagulation dysfunction, portal hypertension, obvious varicose veins or ruptured bleeding in the esophageal and gastric fundus, that is, massive upper gastrointestinal bleeding. Most patients die of shock caused by large amount of bleeding.

4. Hepatic encephalopathy, including cerebral edema and brain herniation. Due to severe damage to liver function, the metabolism of some toxins such as ammonia decreases, resulting in brain symptoms.

5. Edema of both lower limbs, ascites, pleural effusion, and even pericardial effusion and spontaneous bacterial peritonitis. Edema occurs when water in the blood seeps into the interstitial space due to a decrease in the liver's ability to synthesize albumin. If the ascitic fluid is contaminated with bacteria, spontaneous bacterial peritonitis will occur, with a very high mortality rate.