Left eye muscle spasms may often be caused by primary eye spasm, and the occurrence of these symptoms may sometimes even seriously affect vision and cause functional blindness. Therefore, when treating it, you must pay attention to the correct use of medication, including physical therapy and surgical treatments. 1. Primary blepharospasm : It is caused by spasmodic contraction of the orbicularis oculi muscle, resulting in involuntary closure of the eyelids. It is often a bilateral lesion with progressive progression. 2/3 are women, and most develop the disease over the age of 60. The cause is unknown. The frequency and duration of spasms vary. In mild cases, the orbicularis oculi muscle has paroxysmal, frequent small twitches that do not affect eye opening. In severe cases, the twitching is obvious, making it difficult to open the eyes, affecting vision and causing functional blindness. In most patients, symptoms stabilize within 3 to 5 years. One third of patients have associated movement disorders such as Meige syndrome, essential tremor, or Parkinson's disease. Secondary blepharospasm caused by keratoconjunctivitis, trichiasis, and blepharitis should be excluded during diagnosis. 2. The drug and physical treatments for this disease include: clonazepam, Antan, acupuncture, transcutaneous facial nerve thermolysis, etc., but all have little effect. Surgical treatments include: myectomy of the orbicularis oculi and supercilii muscles combined with browplasty and palpebrae superioris muscle reinforcement, and selective facial nerve extraction combined with muscle stripping. However, the effects of these methods are not ideal. The side effects of the former include: forehead numbness and eyelid edema, and the latter may have serious complications of facial nerve paralysis, manifested as ptosis, lagophthalmos, corneal exposure, and ectropion. 50% of patients relapse after surgery. 3. Hemifacial spasm: It is a unilateral lesion involving periodic tonic contraction of the facial muscles. Spasms usually begin in the orbicularis oculi muscle and gradually spread to other parts of the area and can occur during sleep or wakefulness. It usually starts in middle age and is more common in women. May be accompanied by unilateral facial muscle weakness. The cause is usually compression of the seventh cranial nerve root by vascular structures or a tumor at the cerebellopontine angle. 4. Vascular lesions account for 90% of cases, and less than 1% of cases are due to posterior cranial fossa tumors. Drug treatment includes carbamazepine, diazepam, phenytoin sodium, etc., as well as biofeedback, and surgical treatment includes muscle resection. Selective facial nerve resection. However, complications such as hearing loss, otitis media, and cerebrospinal fluid leakage may occur. |
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